Because of a lapse in government funding, the information on this website may not be up to date, transactions submitted via the website may not be processed, and the agency may not be able to respond to inquiries until appropriations are enacted.
The NIH Clinical Center (the research hospital of NIH) is open. For more details about its operating status, please visit cc.nih.gov.
Updates regarding government operating status and resumption of normal operations can be found at USA.gov.

 

Summary: A team of scientists examining the mechanisms of neuropathy, or chronic pain due to nerve damage, after spinal cord injury (SCI) have identified a new role for a protein in the mouse nervous system that is associated with post-SCI pain, suggesting a new cellular target for developing and testing treatments for neuropathy. 

Wu J, Renn CL, Faden AI, Dorsey SG. 2013. TrkB.T1 Contributes to Neuropathic Pain after Spinal Cord Injury through Regulation of Cell Cycle Pathways. J Neurosci. 2013 Jul 24;33(30):12447-63. PMID: 23884949